Trainee-initiated Session

  • Program
  • Trainee-initiated Session
  • Program
  • Application (신청모집)

TIS1. From Understanding to Programming Life: Emerging technologies for Reconstructing Biological Systems

Date May 27, 2026 (WED) Time 11:00-12:40 (KST) Venue 2F 203-204
Supported byy t1-3
Organizer & Chair Minjun KWAK (POSTECH, Korea), Yemin JO (POSTECH, Korea)

A paradigm shift is emerging from analyzing biological systems toward designing and reconstructing them, driven by technologies that enable life engineering across molecular, cellular, and tissue levels. In this session, we discuss how advances in these approaches—such as gene editing, cell reprogramming, organoid platforms, biofabrication, and bio-robotics—are facilitating the experimental redesign and construction of biological systems.

Lecture Time
Speaker

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Lecture Title
20'
Sung Ik Cho KAIST, Korea
From CRISPR to Beyond: The Evolution and Future of Genome Editing
20'
Keel Yong Lee Sejong University, Korea
Engineering Living Systems: From Bottom-Up Design to Translational Biological Systems
20'
Seon-Jin Kim POSTECH, Korea
Tubular Hepatobiliary Tissue Construct Enabling Bile Acid Drainage for Treating Pediatric Hepatobiliary Diseases
20'
Samgmin Lee Kyung Hee University, Korea
Cross-species intestinal organoids for comparative vertebrate stem cell Intestinal organoid zoo of 26 species reveals conserved and divergent programs of biology
20'
Minjun Kwak POSTECH, Korea
Remodeling tissue-wide networks to promote repair through mosaic and transient in vivo reprogramming

TIS2. Toward Predictive Metabolism: Multimodal AI, Wearable Sensors, and Lifelog Data for Disease Prediction

Date May 27, 2026 (WED) Time 11:00-12:40 (KST) Venue 2F 205
Supported by t2-4
Organizer & Chair Eun-Seo BAEK (Seoul National University, Korea)
Chair Ja Yil LEE (UNIST, Korea)

Physiological and behavioral data continuously collected from wearable devices in everyday life are becoming an important foundation for AI-based early disease prediction and personalized healthcare. This session highlights recent advances in multimodal AI that integrate such real-world data with omics, metabolic network modeling, biosensing, lifelog data, and clinical records to advance disease prediction and precision medicine. These approaches support digital biomarker discovery, drug-target and response prediction, early cancer detection, and the prediction of metabolic states and disease-associated physiological changes, while expanding the potential of clinically actionable and personalized medical AI. By linking real-world healthcare data with molecular-level insights, this session also reflects how progress in translational medicine is grounded in fundamental research on biological systems and disease mechanisms.

Lecture Time
Speaker

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Lecture Title
25'
Hwiyoung KimHallym University Medical Center, Korea
From Data to Foresight: Generative Multimodal AI for Early Disease Prediction and Precision Medicine
25'
Soo Youn LeeNational Institute of Health, OHTAC, Korea
Generative AI-Driven Analysis of Lifelog Multimodal Data for Health Prediction
20'
Seongmo KangKAIST, Korea
Host Metabolic Rewiring as a Therapeutic Target: A Genome-Scale Metabolic Model for Rapid Antiviral Discovery
15'
Jaehun JeonKAIST, Korea
Wearable Microfluidic Platform for Label-Free Metabolite Profiling in Sweat
15'
Minkyung ChoiSeoul National University, Korea
Predicting Metabolic and Cardiovascular Diseases from Lifestyle Questionnaires: Quantifying the Incremental Value of Clinical Measurement

TIS3. Genome State Rewiring in Cancer: Hidden Drivers of Tumor Evolution and Therapy Resistance

Date May 27, 2026 (WED) Time 12:55-14:35 (KST) Venue 2F 203-204
Supported by t1-3
Organizer & Chair Hyomin LEE (Oscotec, Korea), Haein CHOI (Oscotec, Korea)

This session focuses on therapy-induced adaptive mechanisms that link tumor evolution and therapy resistance. Moving beyond conventional mutation-targeted therapies, it highlights how genome state changes—such as whole-genome doubling (WGD), replication stress, and associated ploidy alterations—drive cancer evolution under therapeutic pressure. These processes are discussed as key mechanisms promoting tumor survival and adaptation. The session further explores genome state changes as emerging therapeutic targets and their potential to overcome therapy resistance.

Lecture Time
Speaker

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Lecture Title
25'
Kyungjae MyungIBS & UNIST, Korea
Investigating and translating for potenital clincial application of molecular mechanisms of genomic integrity.
20'
Yuseong LeeKyung Hee University, Korea
Radiotherapy-induced senescent cells orchestrate a pro-metastatic niche and drive radio-resistance in head and neck cancer
25'
Hee Jin ChoKyungpook National University, Korea
Transcriptomic Dissection of the Tumor Microenvironment to Guide Target Discovery and Response Prediction
30'
Taeyoung YoonOscotec. Inc., Korea
Toward Anti-Cancer Anti-Resistance Therapy

TIS4. The Big Question: What is the Destiny of the 'Wet Lab' in the AI Era?

Date May 27, 2026 (WED) Time 12:55-14:35 (KST) Venue 2F 205
Supported by t24
Organizer & Chair Yulhui SONG (Seoul National University, Korea)
Chair Hanseul YANG (KAIST, Korea)

What is the role of the biologist when AI provides the answers?
Powerful computational tools are rapidly becoming indispensable in the research landscape. This session strongly advocates for their active integration into biological research, while exploring that what AI cannot fill — designing the right questions and validating predictions through experiment — remains the defining role of the wet lab scientist.

Lecture Time
Speaker

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Lecture Title
25'
Sanguk KimPOSTECH, Korea
Human learning vs. machine learning in AI-driven bio-discovery
25'
Jae Bum KimSeoul National University, Korea
Beyond Algorithms: The Essential Role of Wet Lab Biology in Adipose Tissue Research
25'
Jeong-Ryeol GongKAIST, Korea
BENEIN: Development and Validation of a Single-Cell Boolean Network Pipeline for Identifying Master Regulators of Colorectal Cancer Reversion
25'
Hyojin SonKAIST, Korea
AI Needs Biology: Structure-Based Deep Learning for Mechanistic Modeling of Allostery in Ligand-Induced GPCR Activity